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In Vivo Models for Golgi Fragmentation and the Molecular Pathogenesis of Alzheimer’s Disease

2023

The Golgi apparatus is required for normal function of all cells. This proposal is aimed at determining whether reversal of Golgi fragmentation by overexpression of a key Golgi protein (called GRASP55) in the mouse brain can modulate the behavioral, molecular and histological features of two mouse models of Alzheimer’s disease.

Sam Gandy Studies of Alternative Neurodegenerative Pathways Translational Yanzhuang Wang

In Vivo Models for Golgi Fragmentation and the Molecular Pathogenesis of Alzheimer’s Disease

Marc Weinberg, Steven Arnold,

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In Vivo Models for Golgi Fragmentation and the Molecular Pathogenesis of Alzheimer’s Disease

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In Vivo Models for Golgi Fragmentation and the Molecular Pathogenesis of Alzheimer’s Disease

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