March 28 2019
Posted February 15, 2018
CD33 Variant
In a study co-funded by Cure Alzheimer’s Fund and the National Institutes of Mental Health, Rudy Tanzi, Ph.D., showed that excessive amounts of CD33 play a role in the development of late-onset Alzheimer’s disease. A variant of the gene CD33 impedes the clearance of amyloid beta. However, a mutation of the gene causes amyloid to be cleared more quickly and is actually protective against the disease. Tanzi first identified the gene as part of the Alzheimer’s Genome Project™, an effort started by Cure Alzheimer’s Fund in 2008 to identify all genes connected to Alzheimer’s disease.
March 28 2019
March 28 2019
March 28 2019
March 28 2019
March 28 2019
March 28 2019